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This observation is important because the conventional assumption, based on rodents, has been that esterified forms of vitamin E such as a-tocopheryl acetate, a-tocopheryl nicotinate, and a-TS can be absorbed from the intestinal tract only after they are hydrolyzed to a-tocopherol

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Specific metabolic dependencies such as these can potentially be exploited wither through small molecule inhibition or nutrient deprivation