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Yield: 800 mg, 88%

Although iron-dependent prolyl hydroxylases (PHDs) regulate both isoforms and iron chelation can stabilize HIF-1 and HIF-2 in vitro (74), these observations collectively suggest that the gut may implement additional, tissue-specific mechanisms that bias HIF isoform activity and downstream iron handling

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The reaction was initiated by the addition of 90 L of a glucose-6-phosphate dehydrogenase (G6PDH) working solution