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It is anticipated that more clinical trials will focus on the therapeutic potential of nanomaterial-based therapeutic agents, rather than on their monotonous role as drug delivery systems

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The delivered miR-29a specifically targeted the MMP-2 3UTR, reducing protein expression by more than 60% and disrupting a critical fibrotic amplification loop: MMP-2-mediated E-cadherin degradation releases -catenin, which translocates to the nucleus to activate -SMA/fibronectin expression, further stimulating MMP secretion

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