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Mitochondrial damage associated with apoptosis and necrosis can release damage-associated molecular pattern molecules (DAMPs) and histones that activate inflammatory pathways to exacerbate the lung disease ( FIGURE 1 2.1 Mechanisms of mitochondrial dysfunction Mitochondrial dysfunction can arise through a wide array of genetic and environmental factors and has been implicated in a wide variety of pathological states

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Overall, 32% of current smokers at baseline who were part of a sample resurveyed 3 years later had quit [16]

Flavored tobacco products are seen as a gateway for youth who are smoking

Individuals with depressed mood have poorer smoking cessation outcomes