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Previous studies have shown that PI3K/Akt/mTOR and STAT3 are the main orchestrators of the immune response and keratinocyte hyperproliferation that sustains the chronic epidermal hyperplasia [83, 84]

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Increased dosage of a sir-2 gene extends lifespan in Caenorhabditis elegans

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6 Conclusion Oxidative cell death plays a central role in CNS diseases, and addressing its multifaceted nature is crucial for disease management