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Assistant Director for Digital Content Creation and Engagement, Harvard Health Publishing About the Reviewer Megan Noe, MD, MPH, MSCE, Former Editorial Advisory Board Member, Harvard Health Publishing Disclaimer: As a service to our readers, Harvard Health Publishing provides access to our library of archived content

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This evidence raises an intriguing hypothesis: co-treatment with DHAA and erastin or IKE favors the induction of ferroptosis in cancer cells because both molecules are closely related in tumor cell death-induction mechanisms

In this pathway, p62 binds to Keap1 via its Keap1-interacting region (KIR) and recruits it to the autophagosome through the LC3-interacting region (LIR), leading to Keap1 degradation in autolysosomes [29]

Both overexpression of Trx in transgenic mice and injection of Trx into old wild-type mice improved endothelial-dependent relaxation [257]