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The hydroxyproline level was normalized leading to inhibition of interstitial collagen accumulation after treatment with a combination of the 2 drugs, which may indicate their potential antifibrotic effect

The expression levels of immune checkpoint molecules (CD44, TNFSF18 and NRP1) varied across the risk groups (high- and low-)

First, proteins within one group (S-rich, S-poor or HMW) have a similar structure

Paradoxically, recent studies indicate that mTORC1 can activate the adaptive stress-responsive transcription factor ATF4 through mechanisms distinct from its canonical induction by the integrated stress response (ISR)

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