Poor health outcomes in infants with vitamin B 12 deficiency include anemia, abnormal skin and hair development, convulsions, weak muscle tone, failure to thrive, mental developmental delay, and potentially abnormal movements.[4-8] Well-recognized at risk groups are exclusively breastfed infants of mothers with B 12 deficiency due to minimal or no dietary intake of animal products[9-21] or pernicious anemia caused by a maternal malabsorption of B 12 .[18,22] Infant vitamin B 12 status can be improved through maternal B 12 supplementation during pregnancy and lactation.[23-26] Deficient mothers who miss the opportunity to supplement during pregnancy should still be encouraged to supplement during early lactation since infant vitamin B 12 status correlates with milk vitamin B 12 levels in breastfed infants up to 6 months of age.[27-30] Although there are cases reported of exclusively breastfed infants with vitamin B 12 deficiency having biochemical and clinical improvement through adequate maternal supplementation alone,[9] direct supplementation of the infant is recommended when such treatments are available.[31-33] Drug Levels Methylcobalamin is the major form of vitamin B 12 in milk
Glutathione removes toxins, harmful chemicals, and free radicals from the body
There are several different histamine receptors, known as H1, H2, H3, and H4 receptors
"2013 AHA/ACC/TOS guideline for the management of overweight and obesity in adults: A report of the American College of Cardiology/American Heart Association Task Force on Practice Guidelines and The Obesity Society (in Eng)
Two clinical trials investigated the benefits of B12 in non-diabetic neuropathy
