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They mainly trigger apoptosis of CD4 + T cells in vivo ( 86) by fostering and maintaining an anti-inflammatory IL-10 + phenotype, while also slowing down the activation, maturation, and proliferation of CD4 + T cells (87, 88)

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In addition, induced macrophages from PARK2 knockout mice have increased levels of inflammatory cytokines and PARK2 knockout mice have increased susceptibility to inflammation-related DA neuron degeneration 47,48

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The plasma levels of CXCL1 ( K ), CXCL2 ( L ), TNF- ( M ), and IL-10 ( N ) were analyzed by ELISA ( n = 5