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One of the mechanisms proposed for the disease is an immune cell recognition of myelin as being a foreign substance This may be due to inflammation induced changes causing the myelin to look foreign to the immune system, or to changes associated with viral infection

The restitution of redox equilibrium in the face of an oxidative challenge, therefore, requires an adaptive cross-talk between signaling pathways sensing variations in p O 2 and genetically regulated transcription factors [2, 3, 1618, 26, 51]

the expected number of deaths is less than it should be, so the observed risk ratio is overestimated

Evaluation of DNA interaction, genotoxicity and oxidative stress induced by iron oxide nanoparticles both in vitro and in vivo: attenuation by thymoquinone