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To circumvent this problem, we developed an endosome-escaping peptide comprised of histidine-rich sequences (Fig

Shalev et al., 2000

Consistently, in HepG2 hepatocytes, capsaicin activates AMPK, increases PGC-1 abundance, improves mitochondrial function, and suppresses lipogenesis, linking mitochondrial biogenesis to coordinated control of lipid metabolism (10)